Wheel running from a juvenile age delays onset of specific motor deficits but does not alter protein aggregate density in a mouse model of Huntington's disease

10.1186/1471-2202-9-34

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Main Authors: Van Dellen, A, Cordery, P.M, Spires, T.L, Blakemore, C, Hannan, A.J
Other Authors: DUKE-NUS MEDICAL SCHOOL
Format: Article
Published: 2020
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age
Online Access:https://scholarbank.nus.edu.sg/handle/10635/177978
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spelling sg-nus-scholar.10635-1779782023-11-01T08:29:15Z Wheel running from a juvenile age delays onset of specific motor deficits but does not alter protein aggregate density in a mouse model of Huntington's disease Van Dellen, A Cordery, P.M Spires, T.L Blakemore, C Hannan, A.J DUKE-NUS MEDICAL SCHOOL huntingtin HD protein, human nerve protein nuclear protein age animal experiment animal model animal tissue article brain weight cingulate gyrus controlled study corpus striatum environmental enrichment exercise exploratory behavior female habituation Huntington chorea locomotion male motor coordination motor dysfunction mouse neuroprotection nonhuman open field test physical activity protein aggregation rotarod test running sensory stimulation aging animal atrophy brain cell inclusion disease course disease model environmental planning genetics Huntington chorea kinesiotherapy metabolism methodology motor activity pathology pathophysiology transgenic mouse Aging Animals Atrophy Brain Disease Models, Animal Disease Progression Environment Design Exercise Therapy Exploratory Behavior Female Huntington Disease Inclusion Bodies Male Mice Mice, Transgenic Motor Activity Nerve Tissue Proteins Nuclear Proteins Physical Conditioning, Animal 10.1186/1471-2202-9-34 BMC Neuroscience 9 34 2020-10-20T04:43:41Z 2020-10-20T04:43:41Z 2008 Article Van Dellen, A, Cordery, P.M, Spires, T.L, Blakemore, C, Hannan, A.J (2008). Wheel running from a juvenile age delays onset of specific motor deficits but does not alter protein aggregate density in a mouse model of Huntington's disease. BMC Neuroscience 9 : 34. ScholarBank@NUS Repository. https://doi.org/10.1186/1471-2202-9-34 14712202 https://scholarbank.nus.edu.sg/handle/10635/177978 Attribution 4.0 International http://creativecommons.org/licenses/by/4.0/ Unpaywall 20201031
institution National University of Singapore
building NUS Library
continent Asia
country Singapore
Singapore
content_provider NUS Library
collection ScholarBank@NUS
topic huntingtin
HD protein, human
nerve protein
nuclear protein
age
animal experiment
animal model
animal tissue
article
brain weight
cingulate gyrus
controlled study
corpus striatum
environmental enrichment
exercise
exploratory behavior
female
habituation
Huntington chorea
locomotion
male
motor coordination
motor dysfunction
mouse
neuroprotection
nonhuman
open field test
physical activity
protein aggregation
rotarod test
running
sensory stimulation
aging
animal
atrophy
brain
cell inclusion
disease course
disease model
environmental planning
genetics
Huntington chorea
kinesiotherapy
metabolism
methodology
motor activity
pathology
pathophysiology
transgenic mouse
Aging
Animals
Atrophy
Brain
Disease Models, Animal
Disease Progression
Environment Design
Exercise Therapy
Exploratory Behavior
Female
Huntington Disease
Inclusion Bodies
Male
Mice
Mice, Transgenic
Motor Activity
Nerve Tissue Proteins
Nuclear Proteins
Physical Conditioning, Animal
spellingShingle huntingtin
HD protein, human
nerve protein
nuclear protein
age
animal experiment
animal model
animal tissue
article
brain weight
cingulate gyrus
controlled study
corpus striatum
environmental enrichment
exercise
exploratory behavior
female
habituation
Huntington chorea
locomotion
male
motor coordination
motor dysfunction
mouse
neuroprotection
nonhuman
open field test
physical activity
protein aggregation
rotarod test
running
sensory stimulation
aging
animal
atrophy
brain
cell inclusion
disease course
disease model
environmental planning
genetics
Huntington chorea
kinesiotherapy
metabolism
methodology
motor activity
pathology
pathophysiology
transgenic mouse
Aging
Animals
Atrophy
Brain
Disease Models, Animal
Disease Progression
Environment Design
Exercise Therapy
Exploratory Behavior
Female
Huntington Disease
Inclusion Bodies
Male
Mice
Mice, Transgenic
Motor Activity
Nerve Tissue Proteins
Nuclear Proteins
Physical Conditioning, Animal
Van Dellen, A
Cordery, P.M
Spires, T.L
Blakemore, C
Hannan, A.J
Wheel running from a juvenile age delays onset of specific motor deficits but does not alter protein aggregate density in a mouse model of Huntington's disease
description 10.1186/1471-2202-9-34
author2 DUKE-NUS MEDICAL SCHOOL
author_facet DUKE-NUS MEDICAL SCHOOL
Van Dellen, A
Cordery, P.M
Spires, T.L
Blakemore, C
Hannan, A.J
format Article
author Van Dellen, A
Cordery, P.M
Spires, T.L
Blakemore, C
Hannan, A.J
author_sort Van Dellen, A
title Wheel running from a juvenile age delays onset of specific motor deficits but does not alter protein aggregate density in a mouse model of Huntington's disease
title_short Wheel running from a juvenile age delays onset of specific motor deficits but does not alter protein aggregate density in a mouse model of Huntington's disease
title_full Wheel running from a juvenile age delays onset of specific motor deficits but does not alter protein aggregate density in a mouse model of Huntington's disease
title_fullStr Wheel running from a juvenile age delays onset of specific motor deficits but does not alter protein aggregate density in a mouse model of Huntington's disease
title_full_unstemmed Wheel running from a juvenile age delays onset of specific motor deficits but does not alter protein aggregate density in a mouse model of Huntington's disease
title_sort wheel running from a juvenile age delays onset of specific motor deficits but does not alter protein aggregate density in a mouse model of huntington's disease
publishDate 2020
url https://scholarbank.nus.edu.sg/handle/10635/177978
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