Single cardiomyocyte nuclear transcriptomes reveal a lincRNA-regulated de-differentiation and cell cycle stress-response in vivo

10.1038/s41467-017-00319-8

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Main Authors:	See, K, Tan, W.L.W, Lim, E.H, Tiang, Z, Lee, L.T, Li, P.Y.Q, Luu, T.D.A, Ackers-Johnson, M, Foo, R.S
Other Authors:	MEDICINE
Format:	Article
Published:	Nature Publishing Group 2020
Subjects:	alpha actinin 2 CD34 antigen cyclin G1 homeobox protein Nkx-2.5 long untranslated RNA nuclear RNA stem cell antigen 1 vasculotropin receptor 2 GAS5 long non-coding RNA, human NODAL protein, human protein Nodal transcriptome cardiovascular disease cells and cell components genetic analysis genetic differentiation heterogeneity laboratory method RNA rodent adult animal cell animal experiment animal tissue Article cardiac muscle cell Ccnd2 gene cell cycle cell dedifferentiation cell stress cell subpopulation controlled study Dstn gene gene gene expression gene regulatory network gene silencing Hand2 gene heart failure heart left ventricle human human cell human tissue in vivo study male Mef2a gene Mef2c gene mouse Msn gene nonhuman RNA sequence upregulation animal C57BL mouse cytology gene expression regulation genetics metabolism pathophysiology physiological stress Mammalia Animals Cell Cycle Cell Dedifferentiation Gene Expression Regulation Gene Regulatory Networks Heart Failure Humans Male Mice Mice, Inbred C57BL Myocytes, Cardiac Nodal Protein RNA, Long Noncoding Stress, Physiological Transcriptome
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/178594
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Institution:	National University of Singapore

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spelling	sg-nus-scholar.10635-1785942024-05-08T09:51:56Z Single cardiomyocyte nuclear transcriptomes reveal a lincRNA-regulated de-differentiation and cell cycle stress-response in vivo See, K Tan, W.L.W Lim, E.H Tiang, Z Lee, L.T Li, P.Y.Q Luu, T.D.A Ackers-Johnson, M Foo, R.S MEDICINE alpha actinin 2 CD34 antigen cyclin G1 homeobox protein Nkx-2.5 long untranslated RNA nuclear RNA stem cell antigen 1 vasculotropin receptor 2 GAS5 long non-coding RNA, human long untranslated RNA NODAL protein, human protein Nodal transcriptome cardiovascular disease cells and cell components genetic analysis genetic differentiation heterogeneity laboratory method RNA rodent adult animal cell animal experiment animal tissue Article cardiac muscle cell Ccnd2 gene cell cycle cell dedifferentiation cell stress cell subpopulation controlled study Dstn gene gene gene expression gene regulatory network gene silencing Hand2 gene heart failure heart left ventricle human human cell human tissue in vivo study male Mef2a gene Mef2c gene mouse Msn gene nonhuman RNA sequence upregulation animal C57BL mouse cardiac muscle cell cytology gene expression regulation genetics metabolism pathophysiology physiological stress Mammalia Animals Cell Cycle Cell Dedifferentiation Gene Expression Regulation Gene Regulatory Networks Heart Failure Humans Male Mice Mice, Inbred C57BL Myocytes, Cardiac Nodal Protein RNA, Long Noncoding Stress, Physiological Transcriptome 10.1038/s41467-017-00319-8 Nature Communications 8 1 255 2020-10-20T10:28:12Z 2020-10-20T10:28:12Z 2017 Article See, K, Tan, W.L.W, Lim, E.H, Tiang, Z, Lee, L.T, Li, P.Y.Q, Luu, T.D.A, Ackers-Johnson, M, Foo, R.S (2017). Single cardiomyocyte nuclear transcriptomes reveal a lincRNA-regulated de-differentiation and cell cycle stress-response in vivo. Nature Communications 8 (1) : 255. ScholarBank@NUS Repository. https://doi.org/10.1038/s41467-017-00319-8 2041-1723 https://scholarbank.nus.edu.sg/handle/10635/178594 Attribution 4.0 International http://creativecommons.org/licenses/by/4.0/ Nature Publishing Group Unpaywall 20201031
institution	National University of Singapore
building	NUS Library
continent	Asia
country	Singapore Singapore
content_provider	NUS Library
collection	ScholarBank@NUS
topic	alpha actinin 2 CD34 antigen cyclin G1 homeobox protein Nkx-2.5 long untranslated RNA nuclear RNA stem cell antigen 1 vasculotropin receptor 2 GAS5 long non-coding RNA, human long untranslated RNA NODAL protein, human protein Nodal transcriptome cardiovascular disease cells and cell components genetic analysis genetic differentiation heterogeneity laboratory method RNA rodent adult animal cell animal experiment animal tissue Article cardiac muscle cell Ccnd2 gene cell cycle cell dedifferentiation cell stress cell subpopulation controlled study Dstn gene gene gene expression gene regulatory network gene silencing Hand2 gene heart failure heart left ventricle human human cell human tissue in vivo study male Mef2a gene Mef2c gene mouse Msn gene nonhuman RNA sequence upregulation animal C57BL mouse cardiac muscle cell cytology gene expression regulation genetics metabolism pathophysiology physiological stress Mammalia Animals Cell Cycle Cell Dedifferentiation Gene Expression Regulation Gene Regulatory Networks Heart Failure Humans Male Mice Mice, Inbred C57BL Myocytes, Cardiac Nodal Protein RNA, Long Noncoding Stress, Physiological Transcriptome
spellingShingle	alpha actinin 2 CD34 antigen cyclin G1 homeobox protein Nkx-2.5 long untranslated RNA nuclear RNA stem cell antigen 1 vasculotropin receptor 2 GAS5 long non-coding RNA, human long untranslated RNA NODAL protein, human protein Nodal transcriptome cardiovascular disease cells and cell components genetic analysis genetic differentiation heterogeneity laboratory method RNA rodent adult animal cell animal experiment animal tissue Article cardiac muscle cell Ccnd2 gene cell cycle cell dedifferentiation cell stress cell subpopulation controlled study Dstn gene gene gene expression gene regulatory network gene silencing Hand2 gene heart failure heart left ventricle human human cell human tissue in vivo study male Mef2a gene Mef2c gene mouse Msn gene nonhuman RNA sequence upregulation animal C57BL mouse cardiac muscle cell cytology gene expression regulation genetics metabolism pathophysiology physiological stress Mammalia Animals Cell Cycle Cell Dedifferentiation Gene Expression Regulation Gene Regulatory Networks Heart Failure Humans Male Mice Mice, Inbred C57BL Myocytes, Cardiac Nodal Protein RNA, Long Noncoding Stress, Physiological Transcriptome See, K Tan, W.L.W Lim, E.H Tiang, Z Lee, L.T Li, P.Y.Q Luu, T.D.A Ackers-Johnson, M Foo, R.S Single cardiomyocyte nuclear transcriptomes reveal a lincRNA-regulated de-differentiation and cell cycle stress-response in vivo
description	10.1038/s41467-017-00319-8
author2	MEDICINE
author_facet	MEDICINE See, K Tan, W.L.W Lim, E.H Tiang, Z Lee, L.T Li, P.Y.Q Luu, T.D.A Ackers-Johnson, M Foo, R.S
format	Article
author	See, K Tan, W.L.W Lim, E.H Tiang, Z Lee, L.T Li, P.Y.Q Luu, T.D.A Ackers-Johnson, M Foo, R.S
author_sort	See, K
title	Single cardiomyocyte nuclear transcriptomes reveal a lincRNA-regulated de-differentiation and cell cycle stress-response in vivo
title_short	Single cardiomyocyte nuclear transcriptomes reveal a lincRNA-regulated de-differentiation and cell cycle stress-response in vivo
title_full	Single cardiomyocyte nuclear transcriptomes reveal a lincRNA-regulated de-differentiation and cell cycle stress-response in vivo
title_fullStr	Single cardiomyocyte nuclear transcriptomes reveal a lincRNA-regulated de-differentiation and cell cycle stress-response in vivo
title_full_unstemmed	Single cardiomyocyte nuclear transcriptomes reveal a lincRNA-regulated de-differentiation and cell cycle stress-response in vivo
title_sort	single cardiomyocyte nuclear transcriptomes reveal a lincrna-regulated de-differentiation and cell cycle stress-response in vivo
publisher	Nature Publishing Group
publishDate	2020
url	https://scholarbank.nus.edu.sg/handle/10635/178594
_version_	1800914511707766784

Single cardiomyocyte nuclear transcriptomes reveal a lincRNA-regulated de-differentiation and cell cycle stress-response in vivo

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