The Neurogenic Potential of Astrocytes Is Regulated by Inflammatory Signals

• Main Authors: Michelucci, A, Bithell, A, Burney, M.J, Johnston, C.E, Wong, K.-Y, Teng, S.-W, Desai, J, Gumbleton, N, Anderson, G, Stanton, L.W, Williams, B.P, Buckley, N.J
• Other Authors: BIOLOGICAL SCIENCES
• Format: Article
• Published: Humana Press Inc. 2020
• Subjects: peroxisome proliferator activated receptor gamma; protein p53; transcriptome; transforming growth factor beta1; bone morphogenetic protein 4; histone; immunoglobulin enhancer binding protein; tumor necrosis factor; animal cell; animal tissue; Article; astrocyte; cell cycle regulation; cell dedifferentiation; cell function; controlled study; epigenetics; histone modification; in vitro study; inflammation; mouse; nerve cell plasticity; neural stem cell; nonhuman; phenotype; signal transduction; animal; biological model; cell line; cell proliferation; central nervous system; drug effects; gene expression profiling; gene expression regulation; genetic epigenesis; genetics; metabolism; nervous system development; newborn; pathology; promoter region; protein processing; time factor; Animals; Animals, Newborn; Astrocytes; Bone Morphogenetic Protein 4; Cell Dedifferentiation; Cell Line; Cell Proliferation; Central Nervous System; Epigenesis, Genetic; Gene Expression Profiling; Gene Expression Regulation; Histones; Inflammation; Mice; Models, Biological; Neural Stem Cells; Neurogenesis; NF-kappa B; Phenotype; Promoter Regions, Genetic; Protein Processing, Post-Translational; Signal Transduction; Time Factors; Transcriptome; Tumor Necrosis Factor-alpha
• Online Access: https://scholarbank.nus.edu.sg/handle/10635/179296
• Institution: National University of Singapore

10.1007/s12035-015-9296-x