The Neurogenic Potential of Astrocytes Is Regulated by Inflammatory Signals

10.1007/s12035-015-9296-x

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Main Authors:	Michelucci, A, Bithell, A, Burney, M.J, Johnston, C.E, Wong, K.-Y, Teng, S.-W, Desai, J, Gumbleton, N, Anderson, G, Stanton, L.W, Williams, B.P, Buckley, N.J
Other Authors:	BIOLOGICAL SCIENCES
Format:	Article
Published:	Humana Press Inc. 2020
Subjects:	peroxisome proliferator activated receptor gamma protein p53 transcriptome transforming growth factor beta1 bone morphogenetic protein 4 histone immunoglobulin enhancer binding protein tumor necrosis factor animal cell animal tissue Article astrocyte cell cycle regulation cell dedifferentiation cell function controlled study epigenetics histone modification in vitro study inflammation mouse nerve cell plasticity neural stem cell nonhuman phenotype signal transduction animal biological model cell line cell proliferation central nervous system drug effects gene expression profiling gene expression regulation genetic epigenesis genetics metabolism nervous system development newborn pathology promoter region protein processing time factor Animals Animals, Newborn Astrocytes Bone Morphogenetic Protein 4 Cell Dedifferentiation Cell Line Cell Proliferation Central Nervous System Epigenesis, Genetic Gene Expression Profiling Gene Expression Regulation Histones Inflammation Mice Models, Biological Neural Stem Cells Neurogenesis NF-kappa B Phenotype Promoter Regions, Genetic Protein Processing, Post-Translational Signal Transduction Time Factors Transcriptome Tumor Necrosis Factor-alpha
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/179296
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Institution:	National University of Singapore

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spelling	sg-nus-scholar.10635-1792962024-11-10T10:51:22Z The Neurogenic Potential of Astrocytes Is Regulated by Inflammatory Signals Michelucci, A Bithell, A Burney, M.J Johnston, C.E Wong, K.-Y Teng, S.-W Desai, J Gumbleton, N Anderson, G Stanton, L.W Williams, B.P Buckley, N.J BIOLOGICAL SCIENCES peroxisome proliferator activated receptor gamma protein p53 transcriptome transforming growth factor beta1 bone morphogenetic protein 4 histone immunoglobulin enhancer binding protein transcriptome tumor necrosis factor animal cell animal tissue Article astrocyte cell cycle regulation cell dedifferentiation cell function controlled study epigenetics histone modification in vitro study inflammation mouse nerve cell plasticity neural stem cell nonhuman phenotype signal transduction animal astrocyte biological model cell line cell proliferation central nervous system drug effects gene expression profiling gene expression regulation genetic epigenesis genetics metabolism nervous system development newborn pathology promoter region protein processing time factor Animals Animals, Newborn Astrocytes Bone Morphogenetic Protein 4 Cell Dedifferentiation Cell Line Cell Proliferation Central Nervous System Epigenesis, Genetic Gene Expression Profiling Gene Expression Regulation Histones Inflammation Mice Models, Biological Neural Stem Cells Neurogenesis NF-kappa B Phenotype Promoter Regions, Genetic Protein Processing, Post-Translational Signal Transduction Time Factors Transcriptome Tumor Necrosis Factor-alpha 10.1007/s12035-015-9296-x Molecular Neurobiology 53 6 3724-3739 2020-10-23T02:46:21Z 2020-10-23T02:46:21Z 2016 Article Michelucci, A, Bithell, A, Burney, M.J, Johnston, C.E, Wong, K.-Y, Teng, S.-W, Desai, J, Gumbleton, N, Anderson, G, Stanton, L.W, Williams, B.P, Buckley, N.J (2016). The Neurogenic Potential of Astrocytes Is Regulated by Inflammatory Signals. Molecular Neurobiology 53 (6) : 3724-3739. ScholarBank@NUS Repository. https://doi.org/10.1007/s12035-015-9296-x 0893-7648 https://scholarbank.nus.edu.sg/handle/10635/179296 Attribution 4.0 International http://creativecommons.org/licenses/by/4.0/ Humana Press Inc. Unpaywall 20201031
institution	National University of Singapore
building	NUS Library
continent	Asia
country	Singapore Singapore
content_provider	NUS Library
collection	ScholarBank@NUS
topic	peroxisome proliferator activated receptor gamma protein p53 transcriptome transforming growth factor beta1 bone morphogenetic protein 4 histone immunoglobulin enhancer binding protein transcriptome tumor necrosis factor animal cell animal tissue Article astrocyte cell cycle regulation cell dedifferentiation cell function controlled study epigenetics histone modification in vitro study inflammation mouse nerve cell plasticity neural stem cell nonhuman phenotype signal transduction animal astrocyte biological model cell line cell proliferation central nervous system drug effects gene expression profiling gene expression regulation genetic epigenesis genetics metabolism nervous system development newborn pathology promoter region protein processing time factor Animals Animals, Newborn Astrocytes Bone Morphogenetic Protein 4 Cell Dedifferentiation Cell Line Cell Proliferation Central Nervous System Epigenesis, Genetic Gene Expression Profiling Gene Expression Regulation Histones Inflammation Mice Models, Biological Neural Stem Cells Neurogenesis NF-kappa B Phenotype Promoter Regions, Genetic Protein Processing, Post-Translational Signal Transduction Time Factors Transcriptome Tumor Necrosis Factor-alpha
spellingShingle	peroxisome proliferator activated receptor gamma protein p53 transcriptome transforming growth factor beta1 bone morphogenetic protein 4 histone immunoglobulin enhancer binding protein transcriptome tumor necrosis factor animal cell animal tissue Article astrocyte cell cycle regulation cell dedifferentiation cell function controlled study epigenetics histone modification in vitro study inflammation mouse nerve cell plasticity neural stem cell nonhuman phenotype signal transduction animal astrocyte biological model cell line cell proliferation central nervous system drug effects gene expression profiling gene expression regulation genetic epigenesis genetics metabolism nervous system development newborn pathology promoter region protein processing time factor Animals Animals, Newborn Astrocytes Bone Morphogenetic Protein 4 Cell Dedifferentiation Cell Line Cell Proliferation Central Nervous System Epigenesis, Genetic Gene Expression Profiling Gene Expression Regulation Histones Inflammation Mice Models, Biological Neural Stem Cells Neurogenesis NF-kappa B Phenotype Promoter Regions, Genetic Protein Processing, Post-Translational Signal Transduction Time Factors Transcriptome Tumor Necrosis Factor-alpha Michelucci, A Bithell, A Burney, M.J Johnston, C.E Wong, K.-Y Teng, S.-W Desai, J Gumbleton, N Anderson, G Stanton, L.W Williams, B.P Buckley, N.J The Neurogenic Potential of Astrocytes Is Regulated by Inflammatory Signals
description	10.1007/s12035-015-9296-x
author2	BIOLOGICAL SCIENCES
author_facet	BIOLOGICAL SCIENCES Michelucci, A Bithell, A Burney, M.J Johnston, C.E Wong, K.-Y Teng, S.-W Desai, J Gumbleton, N Anderson, G Stanton, L.W Williams, B.P Buckley, N.J
format	Article
author	Michelucci, A Bithell, A Burney, M.J Johnston, C.E Wong, K.-Y Teng, S.-W Desai, J Gumbleton, N Anderson, G Stanton, L.W Williams, B.P Buckley, N.J
author_sort	Michelucci, A
title	The Neurogenic Potential of Astrocytes Is Regulated by Inflammatory Signals
title_short	The Neurogenic Potential of Astrocytes Is Regulated by Inflammatory Signals
title_full	The Neurogenic Potential of Astrocytes Is Regulated by Inflammatory Signals
title_fullStr	The Neurogenic Potential of Astrocytes Is Regulated by Inflammatory Signals
title_full_unstemmed	The Neurogenic Potential of Astrocytes Is Regulated by Inflammatory Signals
title_sort	neurogenic potential of astrocytes is regulated by inflammatory signals
publisher	Humana Press Inc.
publishDate	2020
url	https://scholarbank.nus.edu.sg/handle/10635/179296
_version_	1821215308922224640

The Neurogenic Potential of Astrocytes Is Regulated by Inflammatory Signals

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