Natural killer cells from patients with recombinase-activating gene and non-homologous end joining gene defects comprise a higher frequency of CD56bright NKG2A+++ cells, and yet display increased degranulation and higher perforin content

10.3389/fimmu.2017.00798

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Main Authors:	Dobbs, K\|Tabellini, G, Calzoni, E
Other Authors:	PAEDIATRICS
Format:	Article
Published:	2020
Subjects:	CD16 antigen CD56 antigen CD57 antigen chemokine receptor CXCR1 gamma interferon interleukin 10 interleukin 12 interleukin 18 killer cell immunoglobulin like receptor natural killer cell receptor NKG2A perforin tumor necrosis factor adolescent adult Article child controlled study correlation analysis cytomegalovirus infection degranulation Epstein Barr virus infection female fluorescence activated cell sorting gene gene frequency gene mutation herpes zoster human human cell immunophenotyping infant lymphocyte proliferation lymphocytopenia major clinical study male natural killer cell newborn non homologous end joining gene Omenn syndrome protein expression recombinase activating gene severe combined immunodeficiency T lymphocyte X linked agammaglobulinemia
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/179473
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Institution:	National University of Singapore

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spelling	sg-nus-scholar.10635-1794732024-04-24T06:51:53Z Natural killer cells from patients with recombinase-activating gene and non-homologous end joining gene defects comprise a higher frequency of CD56bright NKG2A+++ cells, and yet display increased degranulation and higher perforin content Dobbs, K\|Tabellini, G Calzoni, E PAEDIATRICS CD16 antigen CD56 antigen CD57 antigen chemokine receptor CXCR1 gamma interferon interleukin 10 interleukin 12 interleukin 18 killer cell immunoglobulin like receptor natural killer cell receptor NKG2A perforin tumor necrosis factor adolescent adult Article child controlled study correlation analysis cytomegalovirus infection degranulation Epstein Barr virus infection female fluorescence activated cell sorting gene gene frequency gene mutation herpes zoster human human cell immunophenotyping infant lymphocyte proliferation lymphocytopenia major clinical study male natural killer cell newborn non homologous end joining gene Omenn syndrome protein expression recombinase activating gene severe combined immunodeficiency T lymphocyte X linked agammaglobulinemia 10.3389/fimmu.2017.00798 Frontiers in Immunology 8 JUL 798 2020-10-23T04:42:33Z 2020-10-23T04:42:33Z 2017 Article Dobbs, K\|Tabellini, G, Calzoni, E (2017). Natural killer cells from patients with recombinase-activating gene and non-homologous end joining gene defects comprise a higher frequency of CD56bright NKG2A+++ cells, and yet display increased degranulation and higher perforin content. Frontiers in Immunology 8 (JUL) : 798. ScholarBank@NUS Repository. https://doi.org/10.3389/fimmu.2017.00798 16643224 https://scholarbank.nus.edu.sg/handle/10635/179473 Attribution 4.0 International http://creativecommons.org/licenses/by/4.0/ Unpaywall 20201031
institution	National University of Singapore
building	NUS Library
continent	Asia
country	Singapore Singapore
content_provider	NUS Library
collection	ScholarBank@NUS
topic	CD16 antigen CD56 antigen CD57 antigen chemokine receptor CXCR1 gamma interferon interleukin 10 interleukin 12 interleukin 18 killer cell immunoglobulin like receptor natural killer cell receptor NKG2A perforin tumor necrosis factor adolescent adult Article child controlled study correlation analysis cytomegalovirus infection degranulation Epstein Barr virus infection female fluorescence activated cell sorting gene gene frequency gene mutation herpes zoster human human cell immunophenotyping infant lymphocyte proliferation lymphocytopenia major clinical study male natural killer cell newborn non homologous end joining gene Omenn syndrome protein expression recombinase activating gene severe combined immunodeficiency T lymphocyte X linked agammaglobulinemia
spellingShingle	CD16 antigen CD56 antigen CD57 antigen chemokine receptor CXCR1 gamma interferon interleukin 10 interleukin 12 interleukin 18 killer cell immunoglobulin like receptor natural killer cell receptor NKG2A perforin tumor necrosis factor adolescent adult Article child controlled study correlation analysis cytomegalovirus infection degranulation Epstein Barr virus infection female fluorescence activated cell sorting gene gene frequency gene mutation herpes zoster human human cell immunophenotyping infant lymphocyte proliferation lymphocytopenia major clinical study male natural killer cell newborn non homologous end joining gene Omenn syndrome protein expression recombinase activating gene severe combined immunodeficiency T lymphocyte X linked agammaglobulinemia Dobbs, K\|Tabellini, G Calzoni, E Natural killer cells from patients with recombinase-activating gene and non-homologous end joining gene defects comprise a higher frequency of CD56bright NKG2A+++ cells, and yet display increased degranulation and higher perforin content
description	10.3389/fimmu.2017.00798
author2	PAEDIATRICS
author_facet	PAEDIATRICS Dobbs, K\|Tabellini, G Calzoni, E
format	Article
author	Dobbs, K\|Tabellini, G Calzoni, E
author_sort	Dobbs, K\|Tabellini, G
title	Natural killer cells from patients with recombinase-activating gene and non-homologous end joining gene defects comprise a higher frequency of CD56bright NKG2A+++ cells, and yet display increased degranulation and higher perforin content
title_short	Natural killer cells from patients with recombinase-activating gene and non-homologous end joining gene defects comprise a higher frequency of CD56bright NKG2A+++ cells, and yet display increased degranulation and higher perforin content
title_full	Natural killer cells from patients with recombinase-activating gene and non-homologous end joining gene defects comprise a higher frequency of CD56bright NKG2A+++ cells, and yet display increased degranulation and higher perforin content
title_fullStr	Natural killer cells from patients with recombinase-activating gene and non-homologous end joining gene defects comprise a higher frequency of CD56bright NKG2A+++ cells, and yet display increased degranulation and higher perforin content
title_full_unstemmed	Natural killer cells from patients with recombinase-activating gene and non-homologous end joining gene defects comprise a higher frequency of CD56bright NKG2A+++ cells, and yet display increased degranulation and higher perforin content
title_sort	natural killer cells from patients with recombinase-activating gene and non-homologous end joining gene defects comprise a higher frequency of cd56bright nkg2a+++ cells, and yet display increased degranulation and higher perforin content
publishDate	2020
url	https://scholarbank.nus.edu.sg/handle/10635/179473
_version_	1800914551678435328

Natural killer cells from patients with recombinase-activating gene and non-homologous end joining gene defects comprise a higher frequency of CD56bright NKG2A+++ cells, and yet display increased degranulation and higher perforin content

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