Natural killer cells from patients with recombinase-activating gene and non-homologous end joining gene defects comprise a higher frequency of CD56bright NKG2A+++ cells, and yet display increased degranulation and higher perforin content

Natural killer cells from patients with recombinase-activating gene and non-homologous end joining gene defects comprise a higher frequency of CD56bright NKG2A+++ cells, and yet display increased degranulation and higher perforin content

10.3389/fimmu.2017.00798

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Bibliographic Details
Main Authors:	Dobbs, K\|Tabellini, G, Calzoni, E
Other Authors:	PAEDIATRICS
Format:	Article
Published:	2020
Subjects:	CD16 antigen CD56 antigen CD57 antigen chemokine receptor CXCR1 gamma interferon interleukin 10 interleukin 12 interleukin 18 killer cell immunoglobulin like receptor natural killer cell receptor NKG2A perforin tumor necrosis factor adolescent adult Article child controlled study correlation analysis cytomegalovirus infection degranulation Epstein Barr virus infection female fluorescence activated cell sorting gene gene frequency gene mutation herpes zoster human human cell immunophenotyping infant lymphocyte proliferation lymphocytopenia major clinical study male natural killer cell newborn non homologous end joining gene Omenn syndrome protein expression recombinase activating gene severe combined immunodeficiency T lymphocyte X linked agammaglobulinemia
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/179473
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Institution:	National University of Singapore

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