Chronic urotensin II receptor antagonist treatment does not alter hypertrophy or fibrosis in a rat model of pressure-overload hypertrophy

Chronic urotensin II receptor antagonist treatment does not alter hypertrophy or fibrosis in a rat model of pressure-overload hypertrophy

Urotensin II (UII) is a potential mediator in the pathogenesis of cardiovascular disease, and inhibition of its actions at the urotensin receptor (UT) has been shown to improve cardiac function and structural changes of the myocardium in a model of myocardial infarction. In this study we utilized a...

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Bibliographic Details
Main Authors: Andrew R. Kompa, Bing H. Wang, Arintaya Phrommintikul, Pei Y. Ho, Darren J. Kelly, David J. Behm, Stephen A. Douglas, Henry Krum
Format: Journal
Published: 2018
Subjects:
Biochemistry, Genetics and Molecular Biology
Neuroscience
Online Access:https://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=78249263484&origin=inward
http://cmuir.cmu.ac.th/jspui/handle/6653943832/50625
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Institution: Chiang Mai University

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https://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=78249263484&origin=inward
http://cmuir.cmu.ac.th/jspui/handle/6653943832/50625

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