Chronically high level of tgfb1a induction causes both hepatocellular carcinoma and cholangiocarcinoma via a dominant Erk pathway in zebrafish
10.18632/oncotarget.20357
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sg-nus-scholar.10635-1738012024-11-14T07:54:12Z Chronically high level of tgfb1a induction causes both hepatocellular carcinoma and cholangiocarcinoma via a dominant Erk pathway in zebrafish Yan, C Yang, Q Shen, H.-M Spitsbergen, J.M Gong, Z PHYSIOLOGY BIOLOGICAL SCIENCES leptin mitogen activated protein kinase Smad protein transforming growth factor beta1 adult animal cell animal experiment animal model animal tissue Article bile duct carcinoma carcinogenesis carcinogenicity cholangiocyte controlled study disease course gene induction gene overexpression hormone response human human tissue larva liver cell liver cell carcinoma long term exposure MAPK signaling molecular pathology nonalcoholic fatty liver nonhuman oncogene protein expression satiety tgfb1a gene transgenic zebrafish 10.18632/oncotarget.20357 Oncotarget 8 44 77096-77109 2020-09-01T00:57:43Z 2020-09-01T00:57:43Z 2017 Article Yan, C, Yang, Q, Shen, H.-M, Spitsbergen, J.M, Gong, Z (2017). Chronically high level of tgfb1a induction causes both hepatocellular carcinoma and cholangiocarcinoma via a dominant Erk pathway in zebrafish. Oncotarget 8 (44) : 77096-77109. ScholarBank@NUS Repository. https://doi.org/10.18632/oncotarget.20357 19492553 https://scholarbank.nus.edu.sg/handle/10635/173801 Unpaywall 20200831 |
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leptin mitogen activated protein kinase Smad protein transforming growth factor beta1 adult animal cell animal experiment animal model animal tissue Article bile duct carcinoma carcinogenesis carcinogenicity cholangiocyte controlled study disease course gene induction gene overexpression hormone response human human tissue larva liver cell liver cell carcinoma long term exposure MAPK signaling molecular pathology nonalcoholic fatty liver nonhuman oncogene protein expression satiety tgfb1a gene transgenic zebrafish |
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leptin mitogen activated protein kinase Smad protein transforming growth factor beta1 adult animal cell animal experiment animal model animal tissue Article bile duct carcinoma carcinogenesis carcinogenicity cholangiocyte controlled study disease course gene induction gene overexpression hormone response human human tissue larva liver cell liver cell carcinoma long term exposure MAPK signaling molecular pathology nonalcoholic fatty liver nonhuman oncogene protein expression satiety tgfb1a gene transgenic zebrafish Yan, C Yang, Q Shen, H.-M Spitsbergen, J.M Gong, Z Chronically high level of tgfb1a induction causes both hepatocellular carcinoma and cholangiocarcinoma via a dominant Erk pathway in zebrafish |
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10.18632/oncotarget.20357 |
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PHYSIOLOGY |
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PHYSIOLOGY Yan, C Yang, Q Shen, H.-M Spitsbergen, J.M Gong, Z |
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Article |
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Yan, C Yang, Q Shen, H.-M Spitsbergen, J.M Gong, Z |
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Yan, C |
title |
Chronically high level of tgfb1a induction causes both hepatocellular carcinoma and cholangiocarcinoma via a dominant Erk pathway in zebrafish |
title_short |
Chronically high level of tgfb1a induction causes both hepatocellular carcinoma and cholangiocarcinoma via a dominant Erk pathway in zebrafish |
title_full |
Chronically high level of tgfb1a induction causes both hepatocellular carcinoma and cholangiocarcinoma via a dominant Erk pathway in zebrafish |
title_fullStr |
Chronically high level of tgfb1a induction causes both hepatocellular carcinoma and cholangiocarcinoma via a dominant Erk pathway in zebrafish |
title_full_unstemmed |
Chronically high level of tgfb1a induction causes both hepatocellular carcinoma and cholangiocarcinoma via a dominant Erk pathway in zebrafish |
title_sort |
chronically high level of tgfb1a induction causes both hepatocellular carcinoma and cholangiocarcinoma via a dominant erk pathway in zebrafish |
publishDate |
2020 |
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https://scholarbank.nus.edu.sg/handle/10635/173801 |
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1821210293884157952 |