Arginylation-dependent neural crest cell migration is essential for mouse development

• Main Authors: Kurosaka S., Leu N.A., Zhang F., Bunte R., Saha S., Wang J., Guo C., He W., Kashina A.
• Other Authors: DUKE-NUS MEDICAL SCHOOL
• Format: Article
• Published: 2019
• Subjects: arginyltransferase; cre recombinase; Wnt1 protein; aminoacyltransferase; arginine; Wnt1 protein, mouse; animal cell; animal experiment; animal model; animal tissue; arginylation; article; behavior disorder; brain development; breathing disorder; cell cycle regulation; cell migration; cell motility; cell motion; chemical modification; controlled study; embryo; gene deletion; growth retardation; in situ hybridization; knockout mouse; lethality; morphogenesis; mouse; neural crest cell; nonhuman; palate disease; pathogenesis; pharynx disease; promoter region; protein protein interaction; signal transduction; skull defect; animal; biological model; bone; cell adhesion; cell culture; coculture; congenital malformation; craniofacial malformation; enzymology; gene inactivation; growth, development and aging; mesoderm; metabolism; mouse mutant; neural crest; newborn; palate; pathology; survival; Mus; Aminoacyltransferases; Animals; Animals, Newborn; Arginine; Bone and Bones; Cell Adhesion; Cell Movement; Cells, Cultured; Coculture Techniques; Craniofacial Abnormalities; Gene Knockout Techniques; Growth and Development; Mesoderm; Mice; Mice, Knockout; Models, Biological; Neural Crest; Palate; Survival Analysis; Wnt1 Protein
• Online Access: https://scholarbank.nus.edu.sg/handle/10635/161666
• Institution: National University of Singapore

10.1371/journal.pgen.1000878