VSL#3 Resets Insulin Signaling and Protects against NASH and Atherosclerosis in a Model of Genetic Dyslipidemia and Intestinal Inflammation

10.1371/journal.pone.0045425

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Main Authors:	Mencarelli A., Cipriani S., Renga B., Bruno A., D'Amore C., Distrutti E., Fiorucci S.
Other Authors:	DEAN'S OFFICE (DUKE-NUS MEDICAL SCHOOL)
Published:	2019
Subjects:	dextran sulfate farnesoid X receptor peroxisome proliferator activated receptor gamma vitamin D receptor VSL3 adipose tissue animal cell animal experiment animal model animal tissue antiinflammatory activity aorta atherosclerosis article controlled study disease severity drug efficacy dyslipidemia enteritis histopathology insulin resistance intestine mucosa permeability liver protection male mesentery mouse nonalcoholic fatty liver nonhuman transactivation treatment duration Animals Apolipoproteins E Atherosclerosis Dyslipidemias Fatty Liver Inflammation Insulin Resistance Intestines Male Mice Mice, Knockout Probiotics Signal Transduction Mus
Online Access:	https://scholarbank.nus.edu.sg/handle/10635/161730
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Institution:	National University of Singapore

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spelling	sg-nus-scholar.10635-1617302021-10-05T22:39:38Z VSL#3 Resets Insulin Signaling and Protects against NASH and Atherosclerosis in a Model of Genetic Dyslipidemia and Intestinal Inflammation Mencarelli A. Cipriani S. Renga B. Bruno A. D'Amore C. Distrutti E. Fiorucci S. DEAN'S OFFICE (DUKE-NUS MEDICAL SCHOOL) DUKE-NUS MEDICAL SCHOOL dextran sulfate farnesoid X receptor peroxisome proliferator activated receptor gamma vitamin D receptor VSL3 adipose tissue animal cell animal experiment animal model animal tissue antiinflammatory activity aorta atherosclerosis article controlled study disease severity drug efficacy dyslipidemia enteritis histopathology insulin resistance intestine mucosa permeability liver protection male mesentery mouse nonalcoholic fatty liver nonhuman transactivation treatment duration Animals Apolipoproteins E Atherosclerosis Dyslipidemias Fatty Liver Inflammation Insulin Resistance Intestines Male Mice Mice, Knockout Probiotics Signal Transduction Mus 10.1371/journal.pone.0045425 PLoS ONE 7 9 e45425 2019-11-07T01:17:26Z 2019-11-07T01:17:26Z 2012 Mencarelli A., Cipriani S., Renga B., Bruno A., D'Amore C., Distrutti E., Fiorucci S. (2012). VSL#3 Resets Insulin Signaling and Protects against NASH and Atherosclerosis in a Model of Genetic Dyslipidemia and Intestinal Inflammation. PLoS ONE 7 (9) : e45425. ScholarBank@NUS Repository. https://doi.org/10.1371/journal.pone.0045425 19326203 https://scholarbank.nus.edu.sg/handle/10635/161730 Unpaywall 20191101
institution	National University of Singapore
building	NUS Library
continent	Asia
country	Singapore Singapore
content_provider	NUS Library
collection	ScholarBank@NUS
topic	dextran sulfate farnesoid X receptor peroxisome proliferator activated receptor gamma vitamin D receptor VSL3 adipose tissue animal cell animal experiment animal model animal tissue antiinflammatory activity aorta atherosclerosis article controlled study disease severity drug efficacy dyslipidemia enteritis histopathology insulin resistance intestine mucosa permeability liver protection male mesentery mouse nonalcoholic fatty liver nonhuman transactivation treatment duration Animals Apolipoproteins E Atherosclerosis Dyslipidemias Fatty Liver Inflammation Insulin Resistance Intestines Male Mice Mice, Knockout Probiotics Signal Transduction Mus
spellingShingle	dextran sulfate farnesoid X receptor peroxisome proliferator activated receptor gamma vitamin D receptor VSL3 adipose tissue animal cell animal experiment animal model animal tissue antiinflammatory activity aorta atherosclerosis article controlled study disease severity drug efficacy dyslipidemia enteritis histopathology insulin resistance intestine mucosa permeability liver protection male mesentery mouse nonalcoholic fatty liver nonhuman transactivation treatment duration Animals Apolipoproteins E Atherosclerosis Dyslipidemias Fatty Liver Inflammation Insulin Resistance Intestines Male Mice Mice, Knockout Probiotics Signal Transduction Mus Mencarelli A. Cipriani S. Renga B. Bruno A. D'Amore C. Distrutti E. Fiorucci S. VSL#3 Resets Insulin Signaling and Protects against NASH and Atherosclerosis in a Model of Genetic Dyslipidemia and Intestinal Inflammation
description	10.1371/journal.pone.0045425
author2	DEAN'S OFFICE (DUKE-NUS MEDICAL SCHOOL)
author_facet	DEAN'S OFFICE (DUKE-NUS MEDICAL SCHOOL) Mencarelli A. Cipriani S. Renga B. Bruno A. D'Amore C. Distrutti E. Fiorucci S.
author	Mencarelli A. Cipriani S. Renga B. Bruno A. D'Amore C. Distrutti E. Fiorucci S.
author_sort	Mencarelli A.
title	VSL#3 Resets Insulin Signaling and Protects against NASH and Atherosclerosis in a Model of Genetic Dyslipidemia and Intestinal Inflammation
title_short	VSL#3 Resets Insulin Signaling and Protects against NASH and Atherosclerosis in a Model of Genetic Dyslipidemia and Intestinal Inflammation
title_full	VSL#3 Resets Insulin Signaling and Protects against NASH and Atherosclerosis in a Model of Genetic Dyslipidemia and Intestinal Inflammation
title_fullStr	VSL#3 Resets Insulin Signaling and Protects against NASH and Atherosclerosis in a Model of Genetic Dyslipidemia and Intestinal Inflammation
title_full_unstemmed	VSL#3 Resets Insulin Signaling and Protects against NASH and Atherosclerosis in a Model of Genetic Dyslipidemia and Intestinal Inflammation
title_sort	vsl#3 resets insulin signaling and protects against nash and atherosclerosis in a model of genetic dyslipidemia and intestinal inflammation
publishDate	2019
url	https://scholarbank.nus.edu.sg/handle/10635/161730
_version_	1713212738659942400

VSL#3 Resets Insulin Signaling and Protects against NASH and Atherosclerosis in a Model of Genetic Dyslipidemia and Intestinal Inflammation

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